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fructose in natural foods such as fruits, the amounts are very low and the
liver can handle it.
But now add in a “fructose” overload environment from processed carb
sources (high-fructose corn syrup and even sugar) and your liver will get
overworked and build up deposits of fat. That doesn't mean that sugar is
okay while high-fructose corn syrup isn't. Regular sugar is sucrose which
when broken down becomes equal parts glucose and fructose, nearly
identical to the breakdown of high-fructose corn syrup.
It is this buildup of fat in the liver (also termed “Non-Alcoholic Fatty Liver”)
that is going to increase insulin resistance and all the issues that come
from that like obesity and increased risk of illness and disease.
“The alarming increase in fructose consumption may be an
important contributor to the epidemic of obesity and insulin resistant
diabetes in both pediatric and adult populations. For thousands of
years, the human diet contained a relatively small amount of
naturally occurring fructose from fruits and other complex foods.
Adaptation of humans to a high glucose/low fructose diet has meant
that hepatic carbohydrate metabolism is designed to actively
metabolize glucose with a limited capacity for metabolizing a small
daily intake of fructose. The increasing application of high fructose
sweeteners over the past few decades has resulted in a
considerable rise in the dietary intake of fructose. A high flux of
fructose to the liver, the main organ capable of metabolizing this
simple carbohydrate, disturbs normal hepatic carbohydrate
metabolism leading to two major consequences; perturbations in
glucose metabolism and glucose uptake pathways, and a
significantly enhanced rate of de novo lipogenesis and TG
synthesis, driven by the high flux of glycerol and acyl portions of TG
molecules coming from fructose catabolism.”
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