Journal of Rehabilitation Medicine 51-7 | Page 7

476 X. Lin et al. cardiorespiratory fitness (35). Active in- dividuals tend to exhibit higher cardiore- spiratory fitness, which is associated with increased telomere length (36). Savela et al. reported that men in the highest physical activity group had the lowest mortality rate (37). Ludlow et al. found that moderate levels of physical activity may have a more protective influence on telomere length than both low and high Fig. 4. Meta-analysis of the association between telomere length and moderate exercise. levels of exercise (34). The inconsistency 95% CI: 95% confidence interval; SD: standard deviation. in our study could be due to the increased breadth of physical activity, genetic di- Sensitivity analyses versity of participants, and sample size. Sensitivity analyses were performed to detect influ- Aerobic exercise is an ordinary physical activity that ential studies. Table SII lists the pooled mean dif- is positively associated with telomere length (20, 33, ferences, with one study at a time removed from the 38). Aerobic exercise can improve maximal oxygen meta-analysis. Exercise was positively associated with uptake, which is positively related to telomere length telomere length regardless of whether any study was through the activation of telomerase (39). Werner et al. removed from the meta-analysis. reported that aerobic endurance subjects (e.g continuous running) exhibited increased telomerase activity and telomere-stabilizing proteins compared with untrained DISCUSSION controls, which contributes to chromosomal stability This meta-analysis found that exercise had a positive and integrity (40).On the other hand, individuals who effect on telomere length. There are several possible engaged in long-term aerobic exercise have significantly reasons for this phenomenon. First, inactivity may re- improved body mass index and decreased low-density duce the expression of telomerase reverse transcriptase lipoprotein values(41), which is beneficial for maintain- and telomeric  repeat-binding  factor  2, which could ing telomere length through the modulation of shelterin lead to decreased activity of telomerase and telomere and telomerase dynamics (42, 43). In the current study, stabilizing protein (27). Secondly, the longer telomere however, subgroup analysis did not find that aerobic lengths associated with increased physical activity le- exercise was positively related to longer telomere length. vels may be explained by the overall diminished burden The source of this discrepancy may be attributed to dif- of oxidative stress and inflammation, such as decreased ferences in the intensity and duration of aerobic exercise, levels of high-sensitivity C-reactive protein, inter- participant genetic diversity, sample size, and physical leukin-6, tumour necrosis factor-аlpha, granulocyte activity measurements. colony-stimulating factor, and F2-isoprostane. These Some studies have reported a positive relationship inflammatory indicators have been found to contribute between longer telomere length and other types of exer- to telomere attrition (28–30). Thirdly, physical inac- cise when subgroup analysis was performed according tivity may increase the risk of insulin resistance and to type of exercise, such as resistance exercise and obesity, which may also accelerate telomere attrition yoga. Resistance exercise is also positively associated (31, 32). Nevertheless, many studies did not report a with telomere length, although there has been only linear relationship between telomere length and inten- one study reporting the effect of resistance training sity of exercise (15, 16, 20, 26, 33). These discordant on telomere length (43). Resistance exercise not only findings may be due to differences in the methods of diminished the burden of oxidative stress and inflam- DNA extraction, telomere measurement, sample size, mation, but was also associated with a higher level of and/or genetic differences. satellite cell recruitment and promoted type II muscle The current study also found that moderate and ro- fibre hyperplasia and hypertrophy (44). In addition, bust exercise had positive effects on telomere length, individuals with sarcopaenia exhibited lower walking although the intensity of exercise across these studies speed and grip strength due to low muscle mass (45, differed. These results were consistent confirmed in 46). Compared with non-sarcopaenia patients, telomere many studies (18, 19, 21, 23–25), while other studies lengths were shorter in older outpatients with sarco- found that the highest, but not the lowest, levels of paenia (47). Resistance exercise has been shown to be activity, had a significant association with telomere effective in improving sarcopaenia status and reducing length (26, 34). This result may be explained by higher the risk of functional decline (48, 49). Yoga, which www.medicaljournals.se/jrm