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showed other viruses besides EV-D68 contribute to acute
flaccid myelitis, but it doesn’t definitively demonstrate the
enterovirus C105 specifically caused the girl’s paralysis. Now
medical staff can be less myopic in only looking for EV-D68
in these cases.
C105 was only recently discovered in 2010 in patients
from Peru and the Republic of Congo. The girl in the study
is the first reported case of the virus in the United States.
C105 having a worldwide presence could be problematic
because these newly detected subspecies show great genetic
variation in the region normally targeted by tests. In other
words, C105 is hard to find with conventional tests. C105
may be contributing to flaccid paralysis and if so it could
pose diagnostic challenges in clinical laboratories. The virus
can go unrecognized because it is relatively new and can be
hard to detect. Some tests miss it due to the virus’s variation
in genetic sequence.
The researchers found either or both EV-D68 and C105
enteroviruses in the respiratory tracts of all the children with
acute flaccid myelitis. Yet so far, the viruses have not been
found in the children’s spinal fluid. This matters because a
virus in the respiratory tract would not necessarily cause
paralysis, since it is not affecting the nervous system. In order
to link these enteroviruses with these cases of paralysis with
certainty, the virus(es) need to be present in the spinal fluid.
The study will be published in the October issue of the
journal Emerging Infectious Diseases.
These newly detected subspecies
show great genetic variation in the
region normally targeted by tests.
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