TOLIVE
HEALTH & WELLBEING
Ulcerative colitis :
Suitable exercise program for 50years + focusing on strength , balance and falls prevention .
Delivered by health professionals and caters for all fitness levels . Walking aids are welcomed
Greater West Exercise Physiology SOUTH WINDSOR Monday 10.30am , Tuesday 10.30am Thursday 10.30am Cost is $ 5 per session Unit 5 / 74 Mileham Street South Windsor
North Richmond Neighbourhood Centre NORTH RICHMOND Friday 9am Cost is $ 5 per session 33 William Street North Richmond
Richmond Uniting Church RICHMOND Wednesday 10.30am Cost is $ 5 per session 25 West Market Street Richmond what exactly is it ?
For information on attending the Move To Live Seniors Exercise program please contact 0404 871 484 or 02 4577 2348
SUFFERING constant BACK PAIN ? Want to ENJOY LIFE again ?
Work & play like you used to after just a few sessions !
Andreas Klein Nutritionist & Remedial Therapist
BSc , Adv Dip Nut Med , DRM
FREE OFFER !
If you ’ re sick of being in constant back pain CALL NOW for your FREE 30min Assessment Session !
Valued at $ 55
I ’ ve personally developed this technique after 16 years experience :
“ After years of severe back pain I ’ m now enjoying golf , fully pain free and even started brick-laying again !” John Hasler , Glenmore Park
“ I ’ d given up really - I ’ d tried so many things . In just a couple of sessions the pain and muscle spasms had gone . I ’ d recommend your work to anyone .” Craig Gibson , Richmond
Remedial Muscle Elongation Therapy targets the cause of chronic back pain and inflammation to really eliminate them . This technique works well for shoulders and knees too !
Call Andreas 0418 166 269 with Andreas Klein
Eighty three and a half thousand Australians suffer from ulcerative colitis ( UC ). Patients ’ initial diagnosis is mostly in two age ranges 15-35 and 55-70 . Prevalence increases with age , with slightly more men than women . Patients may be of any race , but are likely to be white , while Ashkenazi Jews are at highest risk . UC is usually categorised according to the parts of the large bowel involved / inflamed , therefore ,
1 . Ulcerative proctitis – anus and rectum only
2 . Proctosigmoiditis – rectum and sigmoid colon
3 . Left sided colitis – rectum , sigmoid and descending colon 4 . Pancolitis – entire colon The end of the small intestine that meets the colon may also occasionally be inflamed . This is called backwash ileitis .
Symptoms may be limited to rectal bleeding alone but as more of the colon becomes involved symptoms may include fever , abdominal and rectal pain , cramping , and an urgency to defecate without being able to go .
Sufferers have ongoing diarrhoea unrelieved by over-the-counter medications ( often also passing blood and puss ), causing dehydration , weight loss , fatigue , anaemia , vitamin deficiencies , serious malnutrition , and bone loss . Complications include - liver , skin ( erythema nodosum ) eye and joint inflammation , perforated colon , increased colon cancer risk , and in serious cases ( 8-10 per cent of the patients ) toxic megacolon and possible death .
The normal colon wall consists of four main layers . 1 . The mucosa is a one cell thick layer of mucus producing ( and other ) cells that contact the bowel contents , sitting atop a very thin layer of connective tissue ( lamina propria ) filled with different immune cells ( macrophages , plasma cells , and others ). The lamina propria sits on a thin layer of muscle tissue ( muscularis mucosae ).
2 . The submucosa lies immediately underneath the mucosa . It is made of a thicker layer of connective tissue that contains nerves , blood / lymph vessels , and many different immune cells .
3 . The muscularis lies immediately beneath the submucosa . It is a thick musclecell-layer that surrounds the colon and contracts rhythmically to move colon contents .
4 . Serosa is an external layer of connective tissue covering of the colon . UC generally affects the mucosa and submucosa . Only in severe disease is the muscularis involved . There is a sharp border between normal and affected ( ulcerated ) tissue .
In UC the disease process is as follows . Something initiates damage to and death of localised areas of colonic epithelial-lining cells . In an attempt to remedy things , the damaged / dying epithelial cells release distress hormones to activate local immune responses increasing local blood flow , attracting and activating various immune cells . The affected local area becomes red and swollen . If cell deaths continue , the mucosa takes on a finely granular look , and becomes friable ( breaks easily ); normal vascular pattern is lost and eventually scattered haemorrhagic areas appear .
This leads to breakdown of the intestinal epithelial barrier , allows bacteria and other materials to enter tissue which activates immune cells further and perpetuates chronic intestinal inflammation . Finally puss-producing mucosal ulcers , characteristic of severe disease appear in between areas of normal tissue .
The question is , what causes colon lining cell damage / death , and is there anything that can be done to stop this ? We will have a look at this next month .
24 ISSUE 148 // SEPTEMBER 2022 theindependentmagazine . com . au THE HAWKESBURY INDEPENDENT