SAEVA Congress 2018 Proceedings | 12-15 February 2018 | ATKV Goudini Spa
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Myocardial dysfunction in horses with AHS – should we be reconsidering our treatment strategy ?
Hewetson , M . a , Shaik , Tb 1
Department of Clinical Science and Services , The Royal Veterinary College , North Mymms , Herts , AL9 7TA , United Kingdom 2
Department of Companion Animal Clinical Studies , Faculty of Veterinary Science , University of Pretoria , Private Bag X04 , Onderstepoort , 0110 , South Africa
Objectives : African horse sickness virus ( AHSV ) is a lethal arbovirus affecting all equids . Four syndromes have been recognized , although it is likely that they represent points on a continuum of virulence rather than distinct manifestations of AHSV infection per se . Endothelial cells are an important site of viral replication , leading to severe endothelial damage . The increase in vascular permeability leads to extravasation of fluid , and death is usually attributed to pulmonary oedema . The observation that viral replication also occurs in myocardial vessels suggests that cardiogenic pulmonary oedema may play a role in the pathogenesis of this disease , and therefore the aim of this study was to evaluate myocardial function in horses with AHS .
Methods : Three adult horses were infected with AHSV . Cardiac examination was performed prior to infection and at a predetermined clinical end-point . Cardiac examination included determination of cardiac troponin I ( cTnI ), electrocardiography , and echocardiography . All horses were euthanized once they reached the clinical
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