SAEVA Proceedings 2016 | Page 231

  A PRACTICAL APPROACH TO LAMENESS IN THE FOAL Andrew Gray, BVSc Lameness in the foal is a commonly presenting complaint and treating it can be a daunting task for veterinarians not accustomed to dealing with foals. As with adult horses, a systematic approach, including a complete history, signalment and thorough clinical examination, is the starting point. Contrary to what most clients will tell you is the aetiology for lameness, most cases of lameness are not the result of the mare standing on the foal. However, there are a few key differences between lameness in a foal and lameness in an adult horse: The history must take into consideration the medical history and the immunoglobulin transfer status of the foal. An infectious aetiology is common, so it is safe to assume that all lameness in foals is septic in origin until proven otherwise. Factors like concentration of horses, hygiene, housing and herd health status all contribute to potential infection risk. Abnormalities in previous haematology profiles, periods of illness, abnormal parturition, particularly abnormal placentas (“red bag”, placentitis) and patent or infected urachus, are all indicators that the lameness may be septic in origin. The clinical examination is also different from that of the adult horse, as foals are typically unhandled and unpredictable. Foals can be hypersensitive, so comparing responses between limbs helps, and patient, careful palpation of the entire foal starting from the hoof and moving proximally, with particular attention to every joint and physis, noting any swelling, heat or pain on flexion or pressure, is especially important for non-obvious sources of lameness. Dynamic evaluation of the gait is best performed with the foal walking and trotting unrestrained next to the mare in hand on a firm, level surface. Foals typically show more exaggerated gait deficits than adults in response to pain, so isolating the lame limb is not always as challenging. Once the lame limb or limbs and possibly the anatomical location responsible for the lameness have been identified, the primary objective is to determine if it is of an infectious or non-infectious aetiology. The use of basic haematology profiles and inflammatory indicators (Serum amyloid A and Fibrinogen) has become routine practice in lame foals, as a systemic white cell count and inflammatory proteins will often be elevated due to septic foci. These are also used as a yardstick to measure the efficacy of treatment of lame foals resulting from septic foci. Proceedings  of  the  South  African  Equine  Veterinary  Association  Congress  2016   230