ON Chiropractic
and for researchers comparing the
efficacy of various interventions. This
Recommended Reading
is an active area of research in Dr.
Srbely’s lab.
∞∞ What is the cause-effect relationship
OCA. “Osteoarthritis: Understanding pain and treatment.” ON Chiropractic
(Winter 2015): 16-21.
between C-Sens and other
pathologies? C-Sens has been linked
to several conditions, but the causality
has not yet been clearly established.
∞∞ How long does the effect of SMT last
Srbely, JZ. “Spinal manipulative therapy and its role in the prevention,
treatment and management of chronic pain.” JCCA (2012): 56(1).
: Srbely, JZ. “Chiropractic science: A contemporary neurophysiologic
paradigm.” JCCA (2010) 54(3):144-146.
when modulating C-Sens? How does
it compare to other therapies?
∞∞ What is the nature of the relationship
between C-Sens, myofascial trigger
points and myofascial pain syndrome?
This is an active research question.
Derjean, D et al. “Dynamic balance of metabotropic inputs causes dorsal horn
neurons to switch functional states.” Nat Neurosci (2003 Mar) 6(3):274-81.
b Maguire, G. “Myofascial pain syndrome: Current concepts & management.”
RRS Education Research Reviews (2011). Online.
Q Ustinova, EE et al. “Sensitization of pelvic nerve afferents and mast cell
Possibilities for the
Profession
infiltration in the urinary bladder following chronic colonic irritation
W
292(1):F123-F130.
hile more research is needed,
exciting possibilities for
chiropractic science and
practice are emerging.
Consider a patient whose C-Sens
is triggered by something that is
musculoskeletal in nature (e.g., a muscle
injury or chronic joint degeneration).
Resolving this injury may also resolve
a series of secondary issues which
may potentially stem from C-Sens
and neurogenic inflammation. For
chiropractors who have observed this
effect in the clinic, this research may
offer a scientific explanation for these
experiences.
The primary cause of C-Sens can
be non-musculoskeletal in nature (e.g.,
a tumour). While SMT cannot address
these other causes, clinicians may be
able to modulate C-Sens by “closing the
gate” using SMT and other therapies. This
way, clinicians could potentially halt or
mitigate the establishment of secondary
conditions of C-Sens and neurogenic
inflammation.
is mediated by neuropeptides.” Am J Physiol Renal Physiol (2007)
Srbely, JZ et al. “Dry needle stimulation of myofascial trigger points evokes
segmental anti-nociceptive effects. J Rehabil Med. 2010; 42(5):463-8.
Srbely, JZ. “New trends in the treatment and management of myofascial pain
syndrome.” Curr Pain Headache Rep (2010) 14:346–352.
Srbely, JZ et al. “Capsaicin-induced central sensitization evokes segmental
increases in trigger point sensitivity in humans.” J Pain (2010) 11(7):636-43.
Srbely, JZ et al. “Immediate effects of spinal manipulative therapy on
regional antinociceptive effects in myofascial tissues in healthy young adults.”
J Manipulative Physiol Ther (2013); 36:333–341.
Moayedi, M and Davis, KD. “Theories of pain: From specificity to gate control.”
Journal of Neurophysiology (2012) 109:5-12.
World Health Organization. “Acupuncture: Review and analysis of reports on
controlled clinical trials.” Geneva: World Health Organization (2002). Online:
http://apps.who.int/iris/handle/10665/42414
Srbely, JZ et al. “Stimulation of myfascial trigger points with ultrasound
induces segmental antinociceptive effects: A randomized controlled study.”
Pain (2008) 139(2):260-6.
www.chiropractic.on.ca
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