Louisville Medicine Volume 62, Issue 5 | Page 21

CDC reported prevalence was 1 in 150 children. Today, because of greater awareness with earlier diagnosis, the prevalence is reported to be 1 in 68 children. ASD is found equally in all racial or ethnic groups, but it is five times more common in boys. Nearly 83 percent of children with ASD have a comorbid neurologic or developmental disorder. Some one in ten children with ASD may have psychiatric disabilities. But nearly half of children with ASD have average or above average intelligence, yet because of communication disability, they may originally be diagnosed with intellectual impairment or psychiatric dysfunction. Autism Spectrum Disorder is the phenotypic expression of a pervasive neurologic disorder, thus the etiology is not singular. Spectrum is the key word: actual behavior and life experiences vary considerably. The individual with ASD may be considered “high functioning” i.e. able to integrate into society, learning the areas of communication that are best avoided. Or he may be very withdrawn, needing physical as well as emotional support. Just as with other conditons, there may be a combination of genetic and external factors causing the expression of ASD, including social influences. Genetic conditions such as Fragile X syndrome (ASD, intellectual impairment, and large ears) have a distinct genetic component. ASD is more commonly found in children with tuberous sclerosis or idiopathic seizure disorder than in the general population. On the other hand, in children who are identical twins, there is variable expression of the symptoms of ASD. The disorder is felt to be a life-long condition that impacts the individual’s capacity to communicate orally, interact socially, and manage repetitive behaviors. People with ASD are more likely to have seizure disorders, as ASD is among the more common neurobehavioral comorbidities of children with active epilepsy. Children with ASD are more subject to sleep disorders and gastrointestinal symptoms. Drug therapy does not improve the ASD, but may be appropriate for any secondary medical conditions. While there is no cure for ASD, there are an increasing number of studies that demonstrate early intervention by a multidisciplinary team of family members with pediatricians/family physicians, specialized behavioral health experts, and geneticists may result in overall better outcomes. This team with the family may pursue a “medical path” or a “social path.” Within the Autism community, there are those who define themselves as “people with autism” and those who call them themselves “Autistic people.” The advocacy group, Autism Self-Advocacy Network (autisticadvocacy.org) recognizes the label “autistic person” is a way of self-identification. Advocacy groups then promote the individual’s recognition of the condition and functioning in life with it. This is the social and behavioral response to the label in a fashion similar to advocacy groups for LGBT or ethnic minorities. This allows the individual to feel a part of a virtual community—something that is more difficult for them to achieve in a physical environment. Matt Cottle of Arizona’s Stuttering King Bakery (stutteringkingbakery. com) has developed high success on social media as he has celebrated his label in cooking, just as my patient has in writing. For many individuals this is the better approach to life as an autistic individual. As she told me, “It is a wonderful relief to have a name for a part of yourself that you were not sure what to call. We are proud to be Autistic.” There are others who prefer “people with autism” in recognition that they are people first and that they have a neurologic condition. This carries ASD to the medical model - establish a diagnosis and develop a therapy path. Living here in Philadelphia, I have developed a working relationship with the Genetic Clinics at Geisinger Medical Center in Danville, Pa. and with their counterparts at Children’s Hospital of Philadelphia, where I trained. Both centers recognize that genetic testing for specific conditions such as Fragile X as well as with micro array analysis can achieve as high as 30% percent accuracy in predicting future ASD expression in infants where there is high degree of concern. Families accepting of this medical model and having genetic testing may not fully benefit unless they get their child into a multidisciplinary team approach that includes the family. That team must respect the cultural values of the family but cannot have all of the therapeutics dictated by the family. Studies show that by use of practice guidelines developed through the American College of Medical Genetics, that there can be improvement in clinical outcomes. AS SHE TOLD ME, “IT IS A WONDERFUL RELIEF TO HAVE A NAME FOR A PART OF YOURSELF THAT YOU WERE NOT SURE WHAT TO CALL. WE ARE PROUD TO BE AUTISTIC.” As a primary care physician, the early role is gaining trust so as to better have the conversation with the patient with ASD. What are their goals? Do they prefer autonomy, and the label of autistic person? If so, then guiding them toward support or affinity groups may be more helpful from a social perspective, leaving the physician to manage any of the neurologic or other medical comorbidities. On the other hand, the individual who adopts the medical model may need more direction from the physician in terms of accurate diagnosis and direction into a treatment plan. With most conditions a physician manages there is not the option of moving primarily down