during the winter . As professional skin enthusiasts , it is a primary responsibility to confidently educate the misinformed and inquiring public on the importance of SPF and to then provide appropriate recommendations . Every year as autumn wanes and winter approaches , we find ourselves facing challenging conversations as to why SPF habits must be maintained . At iS Clinical , we provide consumers worldwide with superior SPF options , but we also make it our mission to support our product usage with concrete evidence by showing its importance and performance . It is necessary to understand why our SPF products should be routinely purchased and applied no differently than they would be in the spring and summer .
Skin is a highly sophisticated organ that is designed to protect and maintain normal bodily functions and block foreign substances . Our skin is constantly renewing itself to act as an adequate shield . In-between the time of mitosis and desquamation , our keratinocyte cells release a complex blend of water-soluble chemicals , antioxidant-rich lipids , and strengthening proteins . This constructs a durable physical and chemical-based acidic barrier , designed to preserve our water and lipid levels and work in tandem with our immune system . UVR is a key antagonist leading this process awry , and UVR is not inactive during the winter !
Ninety five percent of radiation comes from UVA ( ultraviolet aging rays ). UVA penetrates the dermis , deteriorating skin ’ s scaffolding of proteins , such as collagen , elastin , and hyaluronic acid . UVA does not lessen in strength during the winter and is still active and present on the greyest and coldest days . UVA ’ s counterpart , UVB ( ultraviolet burning rays ), are equally damaging . UVB primarily affects the epidermal layers and is directly linked to skin cancer . UVB particularly affects melanocyte , Langerhans , and Merkel cell destruction . They are harmful even without tanning or burning . Believe it or not , there is no such thing as a healthy tan . Tanned and sunburned skin experiences apoptosis – the process of mutated
Our skin is constantly renewing itself to act as an adequate shield .
cells opting for suicide and peeling off . Remaining cells with mutated DNA proceed to birth a permanent chain of mutated cells during mitosis . It is crucial to understand that UVA and UVB are both cytotoxic , mutagenic , and carcinogenic . Both rays contribute to broken capillaries , edema , and erythema . Additionally , they are responsible for excessive TEWL ( transepidermal water loss ) and loss of critical barrier lipids . We expect skin to appear tight and dry after prolonged periods of sun exposure , but the skin naturally experiences water and lipid loss in the winter due to lower temperatures and lack of humidity .
Did you know that even though UVB rays are stronger during summer , you can still get a sunburn during the winter ? This is common for winter sport lovers .
UVA penetrates the dermis , deteriorating skin ’ s scaffolding of proteins , such as collagen , elastin , and hyaluronic acid .
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