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Exercise may help overcome bad genes, aging and help response to therapy
with Andreas Klein
Research shows your genes don’ t determine the person you become. Diet and exercise, have huge positive influences even if you have poor genetics. Illustrating this,‘ mtDNAmutator’ mice have‘ really-bad’ genes. They accumulate 3-5 times as many mitochondrial DNA mutations as wild type mice in equivalent time. This leads to:
• mitochondrial dysfunction( in biogenesis & fission),
• excessive free radical production
• oxidative stress & damage to cells
• accelerated body cell death rates
• loss of cell reserve( aging). This loss of cell reserve( stem cells) leads these mice to suffer from‘ progeria’, a disorder with profound premature aging and early death. Like old people these mice have reduced fertility, muscle loss( sarcopenia) and subcutaneous fat loss, hair loss, curved spines, osteoporosis, anaemia, heart enlargement, and they get all these things very early in life.
Key is that their mitochondria malfunction first, well before the appearance of the actual signs of aging.
Experiments by Safdar etal in 2011 using‘ mtDNA-mutator’ mice, demonstrated how regular endurance exercise can help even these genetically challenged mice. They showed‘ that 5
months of endurance exercise 1. induced systemic mitochondrial biogenesis, 2. prevented mitochondrial DNA deletions and mutations, 3. increased mitochondrial energy production capacity, 4. restored mitochondrial structure, and 5. blunted pathological levels of’ cell death‘ in multiple tissues of the mtDNA mutator mice’.
Endurance exercise conferred complete protection from progeria,‘ multisystem pathology, and prevented premature mortality in these mice’.
Thus, regular endurance exercise was able to overcome the severe genetic defects that these mice had. The authors concluded that,‘ the systemic mitochondrial rejuvenation through endurance exercise promises to be an effective therapeutic approach to mitigating mitochondrial dysfunction in aging and related comorbidities’( i. e. diseases).
Most of us will not have to overcome such severe genetic defects. For many of us, our lack of exercise will be a key contributor to the mitochondrial dysfunction that occurs in our cells and tissues, well before we see the outer signs of aging, including muscle loss.
Appropriate regular endurance exercise seems to be one key preventer of age-related decline, and by keeping you physiologically younger, it may also accelerate your response to remedial therapy.
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20 ISSUE 190 // MARCH 2026 theindependentmagazine. com. au THE HAWKESBURY INDEPENDENT