HEALTH & WELLBEING
Back Inflammation , many people have it but how does it occur ?
SUFFERING constant BACK PAIN ? Want to ENJOY LIFE again ?
Work & play like you used to after just a few sessions !
I ’ ve personally developed this technique after 16 years experience :
“ After years of severe back pain I ’ m now enjoying golf , fully pain free and even started brick-laying again !” John Hasler , Glenmore Park
Andreas Klein Nutritionist & Remedial Therapist
BSc , Adv Dip Nut Med , DRM
“ I ’ d given up really - I ’ d tried so many things . In just a couple of sessions the pain and muscle spasms had gone . I ’ d recommend your work to anyone .” Craig Gibson , Richmond
FREE OFFER !
If you ’ re sick of being in constant back pain CALL NOW for your FREE 30min Assessment Session !
Valued at $ 55
Remedial Muscle Elongation Therapy targets the cause of chronic back pain and inflammation to really eliminate them . This technique works well for shoulders and knees too !
with Andreas Klein
Many clinic patients present with back inflammation and pain . Often , they ’ re taking non-steroidal anti-inflammatory drugs ( NSAIDs ) such as ;
· ibuprofen - Nurofen ,
· diclofenac – Voltaren ,
· naproxen – Naprosyn ,
· celecoxib – Celebrex
These drugs will alleviate back inflammation / pain short term ( a few days ) but seem to be ineffective longer-term . Indeed , long-term intake may produce negative effects .
Inflammation has been around for a long time . It was defined by Roman physician Celsus as constituting a combination of five cardinal signs : 1 . heat ( Calor ) 2 . redness ( Rubor ) 3 . swelling ( Tumour ) 4 . pain ( Dolor ) 5 . loss of function ( Function laesa ) Many people with back pain may relate to these . How inflammation causes pain . Inflammation is a beautifully intricate protective mechanism , designed as the body ’ s response to tissue damage from physical / chemical trauma , or infection . It is a process with distinct stages . At the microscopic level : 1 . physical or chemical trauma , or microbes damage local tissues and cells .
2 . damaged cells make and secrete chemical mediators to notify surrounding cells there ’ s a problem
3 . surrounding immune , nerve and other tissue cells pick up this signal then release additional chemicals to : a . bring extra immune cells from the blood and lymphatic vessels b . stimulate bone marrow to make and release new immune cells c . hypersensitise local nerve cells to heighten pain
4 . immune cells home in on the damaged area , releasing : a . more chemicals of alarm plus , b . huge amounts of peroxide , and free radicals to kill any microbes , c . enzymes to digest damaged tissue and dead microbes d . antibodies to clean-up dead and dying microbes , body-cells , and tissue fragments
Some of the chemical mediators released by cells after damage include : a . Histamine b . Bradykinin and CGRP peptides c . Cytokines ( protein hormones produced by tissues and immune system – e . g . Interleukin-1β & TNFα , substance-P ( for pain ), etc , d . Oxygen and nitrogen free radicals , peroxide , e . Leukotrienes and prostaglandins ( fish-oil based hormones ) f . Metalloproteinase enzymes Note : NSAID ’ s can only modulate production of prostaglandins and leukotrienes - none of the others .
In response to release of all the above chemical mediators :
1 . local blood vessels widen , increasing blood flow and warming the damaged area and become leaky increasing fluid flow from blood vessels into and tissues 2 . local pain receptors are stimulated increasing perceived pain
One reason people have chronic back pain is that the chemical mediators of inflammation that were released acutely after injury have never been cleared from the local tissue . A key goal of muscle elongation therapy as practiced at the clinic is to physically reduce the amount inflammatory chemicals remaining in tissue after an injury . This helps to de-inflame the tissue , reducing / eliminating accompanying pain , and the requirement for medication .
Call Andreas 0418 166 269
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THE HAWKESBURY INDEPENDENT theindependentmagazine . com . au ISSUE 174 // NOVEMBER 2024 27