Australian Doctor 1st September 2023 AD 1st Sept Issue | Page 30

30 HOW TO TREAT : IRRITABLE BOWEL SYNDROME

30 HOW TO TREAT : IRRITABLE BOWEL SYNDROME

1 SEPTEMBER 2023 ausdoc . com . au
CC-BY-SA-3.0 Kyle Thompson , 2006.7
Table 1 . High and low FODMAP foods
Bread and cereals
High FODMAP foods
Wheat / rye / barley-based breads , breakfast cereals , biscuits and snacks
Low FODMAP alternatives
Corn flakes , oats , quinoa flakes , quinoa / rice / corn pasta , rice cakes ( plain ), sourdough spelt bread , wheat / rye / barley-free breads
Dairy and alternatives
Cow ’ s milk , custard , evaporated milk , ice cream , soy milk ( made from whole soybeans ), sweetened condensed milk , yoghurt
Almond milk , brie / camembert cheese , feta cheese , hard cheeses , lactose-free milk , soy milk ( made from soy protein )
Fruits Apples , apple juice , cherries , dried fruit , mango , nectarines , peaches , pears , plums , watermelon
Rock melon , green kiwi fruit , mandarin , orange , pineapple
Nuts and seeds
Cashews , pistachios
Macadamias , peanuts ,
pumpkin seeds / pepitas ,
walnuts
Protein sources
Sugars , sweeteners and confectionery
Most legumes / pulses , some marinated meats / poultry / seafood , some processed meats
High fructose corn syrup , honey , sugar-free confectionery
Eggs , firm tofu , plain cooked meats / poultry / seafood , tempeh
Dark chocolate , maple syrup , rice malt syrup , table sugar
Vegetables Artichoke , asparagus , cauliflower , garlic , green peas , mushrooms , onion , sugar snap peas
Eggplant , beans ( green ), bok choy , green capsicum , carrot , cucumber , lettuce , potato , zucchini
Source : Based on Monash University https :// bit . ly / 2oa3kdV
disorder . 17 These disorders , which
results in IBS , their outcomes sup-
involve inappropriate defecatory
port the implication of gut microbi-
Figure 1 . Bristol stool chart .
mechanics and behaviours , are known by various names , including pelvic floor dyssynergia , anismus or
ome changes contributing to IBS . 20 There is inadequate evidence that excessive bacterial populations in
obstructed defecation .
the small intestine ( also known
There is a wide range of symp-
as small intestinal bacterial over-
toms that may indicate a defecatory
growth [ SIBO ]) cause IBS . It is more
disorder . Patients can complain of
likely that other gut-microbiota
“ constipation ” but on careful ques-
pathways explain IBS symptoms .
tioning this may involve symptoms of incomplete emptying and strain-
Untreatable causes of IBS
ing to pass a stool . These symp-
GUT IMMUNE DYSFUNCTION
toms may indicate a poorly relaxing
Gut immunological pathways ,
anal sphincter or pelvic floor . Some
such as mast cell activation , have
patients paradoxically contract their sphincter and pelvic floor when attempting to defecate , which
been associated with symptoms of IBS . 21 High-quality clinical trials of drugs addressing gut immunolog-
results in an obstructed pattern of
ical targets are lacking , and there-
defecation .
fore these pathways are of unclear
In contrast , others may experi-
significance .
ence a heightened perception of stool entering the rectum and inappropriately defecate earlier than required .
EPIDEMIOLOGY
FUNCTIONAL gut symptoms are
Patients may describe the passage
highly prevalent in the commu-
Figure 2 . Subtypes of IBS based on stool form .
of small frequent stools or excessively feeling the need to defecate
nity . Australian data suggests that 36 % of the general population have
and not producing stool . Associated
symptoms of a functional gastroin-
and laboratory studies have found
patients who experience anxiety , stress or a history of trauma are more
emptying and stimulation of colonic motor function noted . 12
A dramatic study in the 1950s eval-
minor gastrointestinal symptoms . 15 This personality profile can lead to maladaptive behaviours , such as
symptoms of a pelvic floor disorder include frequent or difficult urination and dyspareunia . These mala-
testinal disorder , with 9 % having IBS . 4 Some studies have suggested that functional gastrointestinal
likely to perceive symptoms in the
uated those with and without symp-
overly restrictive diets , in an attempt
daptive defecatory behaviours can
symptoms , which include IBS , are
gastrointestinal tract at a lower symptom threshold . 10 Functional imaging studies ( figure 7 ) have revealed greater , and earlier , activation of pain
toms of IBS , confirming that stress results in changes in motility . Individuals were subjected to pain ( hand in cold water or a head clamp caus-
to control symptoms .
Bidirectional relationship of gut-brain axis
be triggered by specific events such as prior episodes of faecal incontinence , anal fissures ( see figure 8 ) and a history of sexual assault . 18
among the most common presentations to primary care and gastroenterology practice . 22 IBS is more common among younger patients
centres in the brain in patients with IBS compared with healthy controls . 11
ing a headache ) and other emotional stressors while their colonic motility and engorgement was measured . The
In many patients , the experience of gastrointestinal symptoms is distressing , leading to heightened stress
Partly treatable causes of IBS
and women , with the exception of patients from the Indian subcontinent , where it is more common
Impact on gastrointestinal motility
author noted disturbances in motility
and anxiety ( gut symptoms affect-
GUT MICROBIOTA
among men . 23
Emotional and psychological states play a significant role in altering gastrointestinal motility . The impact of one ’ s emotional state on gastrointes-
( increased tone associated with hostility and aggression and decreased tone associated with overt behaviour symbolising hopelessness and defeat ). 13
ing the brain ). The increased distress then leads to heightened perception of visceral gut symptoms and altered motility ( brain affecting the gut ). This
The gut microbiota has been implicated in causing symptoms of IBS . Traditionally , gut microbiota changes have been purported to
DIAGNOSIS AND INVESTIGATION
CLINICIANS are encouraged to
tinal function will not come as a sur-
bidirectional relationship of the gut-
explain patients who develop IBS
make a ‘ positive diagnosis ’ of IBS .
prise , as many may recall needing to
Maladaptive and health-seeking
brain axis can lead to a vicious per-
after a gastrointestinal infection ,
This is achieved when a patient
open their bowels more than once ( or with looser stools ) before examinations or public speaking . Multiple clinical and laboratory studies have demonstrated how anxiety and stress alter gastrointestinal func-
behaviours Some patients with IBS demonstrate abnormal health-seeking behaviours . 14 Patients with IBS who present to medical practitioners often demonstrate perfectionistic personality pro-
petuation of symptoms . 16
PELVIC FLOOR AND DEFECATORY DYSFUNCTION An important , yet poorly recognised , contributor to symptoms of
commonly referred to as post-infectious IBS . 19 Clinical trials demonstrating the benefit of certain antibiotics support the idea that the gut microbiome is a driver for symptoms in IBS . Although faecal trans-
presents with , first , characteristic symptoms ( abdominal pain or discomfort relating to a change in bowel habits or stool form ), and second , an absence of red flags . 24 The discomfort and pain may be either
tion in IBS , with inhibition of gastric
files and may have poor tolerance of
IBS is patients with a defecatory
plant trials have yielded conflicting
relieved or aggravated by a bowel