Acta Dermato-Venereologica 99-7CompleteContent | Page 18
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SHORT COMMUNICATION
Localized Pegfilgrastim-induced Neutrophilic Dermatosis with Tissue G-CSF Expression: A Mimicker
of Sweet’s Syndrome
Toshio ICHIKI 1 , Kazunari SUGITA 1 , Hiroyuki GOTO 1 , Masahisa SHINDO 2 and Osamu YAMAMOTO 1
1
Department of Medicine of Sensory and Motor Organs, Division of Dermatology, Faculty of Medicine, Tottori University, Tottori, and 2 Department
of Dermatology, National Hospital Organization Hamada Medical Center, Hamada, Japan. E-mail: [email protected]
Accepted Mar 8, 2019; E-published Mar 8, 2019
Pegfilgrastim is a granulocyte-colony stimulating factor
(G-CSF) that has recently been developed by using the
pegylation technology, whereby a polyethylene glycol
moiety is attached to filgrastim. Although filgrastim-
induced Sweet’s syndrome has been often reported,
pegfilgrastim-induced Sweet’s syndrome is rare. Here
we report an additional case of neutrophilic dermatosis
induced by pegfilgrastim. Moreover, we have confirmed,
for the first time, the local expression of G-CSF and we
discuss the pathophysiology of the lesion.
CASE REPORT
A 59-year-old woman visited us for an evaluation of a red plaque
on her right ankle (Fig. 1). She had been diagnosed with breast
cancer (cT4bN3aM1, Stag4) at the age of 56 and had been re-
peatedly treated with adjuvant chemotherapy and lenograstim (a
conventional G-CSF) after a right mastectomy with a right axillary
lymphadenectomy. To decrease the frequency of injections, leno-
grastime was replaced by pegfilgrastim. Ten days after the initial
pegfilgrastim administration, she observed an eruption on her left
ankle. Physical examination revealed a red plaque with a strong
induration of the outer side of the right ankle, partly covered with
crusts and scales (Fig. 1). Her body temperature was 37.1°C, and
she had no palpable lymph nodes. Pertinent laboratory findings
included a white blood cell (WBC) count of 18.0 × 10 3 /µl with 91%
neutrophils and 6% lymphocytes. C-reactive protein level was 2.77
mg/dl. Histopathologically, the biopsy specimen showed lobular
neutrophilic panniculitis, without fat necrosis and a lack of nodular
granulomatous phlebitis, and a dense mixed cellular infiltration,
mainly composed of polymorphonulear leukocytes in the deep
dermis and subcutis (Fig. 2a, b). There was also an aggregation
of histiocytes (Fig. 2c). Immunohistochemically, G-CSF was
detected in the cytoplasm of the infiltrating histiocytes (Fig. 2d).
Recently, diagnostic criteria for the drug-induced Sweet’s syn-
drome have been established, separately from those for the classic
Sweet’s syndrome (1). According to these criteria, we could not
make a diagnosis of the drug-induced Sweet’s syndrome because
she was afebrile. Other conditions, including clinical findings,
underlying cause and clinical course, fulfilled the criteria. The
differential diagnosis should include erythema nodosum and
erythema induratum. However, the present case shows a pre-
dominantly lobular panniculitis and a lack of a granulomatous
vasculitis of arterioles, lack of fat necrosis, and a lack of nodular
granulomatous phlebitis exclude these diagnoses (2–4).
Finally, we made a diagnosis of subcutaneous neutrophilic
dermatosis comparable to Sweet’s syndrome. The lesion sponta-
neously and rapidly cleared within 2 weeks without any special
treatment except for the cessation of pegfilgrastim. Instead, treat-
ment with lenograstim was restarted and similar symptoms have
not been observed for over 1 year.
DISCUSSION
We have presented a case of subcutaneous neutrophilic
dermatosis comparable to Sweet’s syndrome, induced
by pegfilgrastim and the existence of G-CSF in the tis-
sue. G-CSF-induced neutrophilic dermatoses, including
Sweet’s syndrome, have been often reported (5). How
ever, to the best of our knowledge, only 3 cases of Sweet’s
syndrome (6–8) and one case of pyoderma gangrenosum
(9) induced by pegfilgrastim have been reported so
Fig. 1. A red plaque with
strong induration of the
outer side of the right ankle
partly covered with crusts
and scales.
This is an open access article under the CC BY-NC license. www.medicaljournals.se/acta
Journal Compilation © 2019 Acta Dermato-Venereologica.
doi: 10.2340/00015555-3170
Acta Derm Venereol 2019; 99: 685–686