Acta Dermato-Venereologica 99-3CompleteContent | Page 27
SHORT COMMUNICATION
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Tazarotene 0.015% Cream as a Potential Topical Agent for Management of Ichthyosis in Dorfman-
Chanarin Syndrome
Luana NICULESCU 1 , Cristel RUINI 1 , Jerome SROUR 1 , Suzanna SALZER 1 , Ines SCHÖNBUCHNER 2 , Tanja VON BRAUNMÜHL 1 ,
Thomas RUZICKA 1 , Ulrich HOHENLEUTNER 3 , Kathrin A. GIEHL 1 , Judith FISCHER 4 and Andreas WOLLENBERG 1 *
Department of Dermatology and Allergy, Ludwig Maximilian University, Frauenlobstr. 9-11, DE-80337 Munich, 2 Department of Human
Genetics and 3 Department of Dermatology, University Medical Center Regensburg, Regensburg, and 4 Department of Human Genetics, Medical
Center – University of Freiburg, Faculty of Medicine, University of Freiburg, Germany. *E-mail: [email protected]
1
Accepted Nov 20, 2018; E-published Nov 21, 2018
Dorfman-Chanarin syndrome (DCS), known as neutral
lipid storage disease with ichthyosis, is an autosomal re-
cessive disorder caused by mutations in the ABHD5 gene
responsible for triglyceride degradation (1, 2). Lipid dro-
plets accumulate in various cells, including granulocytes,
keratinocytes, hepatocytes, Schwann cells and epidermal
Langerhans cells (3). Currently, there is no treatment for
DCS and the management of symptoms and potential
complications requires multidisciplinary coordination.
Skin involvement is characterized by ichthyosiform
non-bullous erythroderma, and adequate treatment poses
a challenge to dermatologists. Various topical therapies,
including emollients and keratolytic agents, have been
proposed to improve ichthyosis, with mostly unsatisfac-
tory results (4). While systemic therapy with retinoids
has been used successfully in patients with ichthyosis,
co-morbidities limit its use in DCS. We report here overall
clinical improvement with local use of tazarotene 0.015%
cream in a female patient with DCS manifesting with
ichthyosis, hepatomegaly and cataracts.
CASE REPORT
An 18-year-old woman born to a consanguineous Kurdish family
presented with a history of generalized congenital ichthyosis.
Previous genetic testing revealed the homozygous mutation
c.594dupC (p.Arg199Glnfs*11) in the ABHD5 gene, consistent
with DCS (1). Furthermore, pathognomonic lipid droplets were
identified in granulocytes by lipid-specific blood smear coloration.
Topical therapy was initiated with tazarotene 0.015% cream
(15 g tazarotene 0.1%, Zorac ®, EMRA-MED Arzneimittel GmbH
(Trittau, Germany), diluted in 100 g cream base) on the face and
left side of the body once daily. The patient was informed about
the teratogenic potential of retinoids, and contraception during
treatment was advised. As an internal control, the other half of the
patient’s body was treated with emollient containing 10% urea in
the same vehicle as the diluted tazarotene cream. The treatment
was not blinded. Blood samples were taken prior to and 8 weeks
after treatment. The liver enzymes, which were elevated prior to
therapy, remained stable with no further increase.
After 8 weeks, a clear delineation of the side treated with
tazarotene 0.015% was evident. The left side had significantly
less scaling than the right side treated with 10% urea, where skin
findings were similar to baseline, and the patient observed sweating
on the left side of her body (Fig. 1e).
To assess these findings, transepidermal water loss (TEWL)
was measured using TEWAMETER TM210 ® and stratum cor-
neum hydration (SCH) using Corneometer ® 825 at baseline and
8 weeks after therapy. An increase in TEWL is seen in patients
with a disrupted skin barrier, indicating increased water loss and
decreased hydration of stratum corneum (4). The side treated with
tazarotene showed lower TEWL and higher SCH values than the
side treated with urea 10%, indicating overall improvement in
skin barrier function following tazarotene treatment (Table SI 1 ).
Optical coherence tomography (OCT), a non-invasive method
to examine skin changes, was performed at baseline and after
8 weeks using VivoSight OCT ® , Firma Michelson Diagnostics
Deutschland GmbH (Schwarzenbruck-Altenthann, Germany),
which allows an imaging depth of approximately 1.5–2 mm (5). A
coherent interpretation of the OCT image was performed by using
histopathological images from a previous biopsy as a template.
At baseline, OCT identified an abnormal hyper-reflective stratum
corneum indicative of hyperkeratosis, an increased epidermal
thickness, a thinned granular layer and papillomatosis, all consis-
tent with the histopathology. Following treatment, OCT showed an
absence of scales, normalization of the epidermal thickness and of
the granular layers (Fig. 1a, b). The epidermal thickness decreased
from 0.2094 ± 0.03560 mm to 0.1508 ± 0.03121 mm after 8 weeks
of therapy (p < 0.0001) (6).
After 8 weeks, urea 10% was stopped and tazarotene 0.015%
cream was applied to the whole body. Follow-up after 13 months
of topical therapy revealed satisfactory clinical improvement.
DISCUSSION
DCS is a disorder with many systemic implications. The
most obvious is physical appearance, since patients have
congenital erythrodermic ichthyosis (7, 8). Keratolytic
agents were not considered as treatment options for our
patient, due to side-effects following systemic absorption.
Although the use of systemic retinoids has been reported
in individual cases of DCS with satisfactory results and
tolerability, concerns regarding worsening of the elevated
liver enzymes precluded its use (7). Tazarotene is a retinoic
acid that has shown favourable results in patients with
ichthyosis when used topically in a 0.1% formulation. We
diluted tazarotene to 0.015% and tested this formulation on
half of the patient’s body. She reported good tolerability,
which allowed for full adherence to therapy.
While many authors have published their experience
with different types of ichthyosis, this is the first report
of topical tazarotene treatment in a patient with DCS (9,
10). Tazarotene is a retinoid that binds selectively to the
RARß and RARγ receptors, mostly expressed in the epi-
dermis. The rapid conversion of the hydrophobic prodrug
tazarotene to the hydrophilic active metabolite tazarotenic
acid ensures a low level of retinoid in the fatty tissue (11).
https://www.medicaljournals.se/acta/content/abstract/10.2340/00015555-3087
1
This is an open access article under the CC BY-NC license. www.medicaljournals.se/acta
Journal Compilation © 2019 Acta Dermato-Venereologica.
doi: 10.2340/00015555-3087
Acta Derm Venereol 2019; 99: 345–346