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SHORT COMMUNICATION
Vasculitis Mimicking Pseudo Erysipelas in Systemic Lupus Erythematosus
Morgane WEISS 1 , Marie-Dominique VIGNON 2 , Clémence LEPELLETIER 1 , Lou MACAUX 1 , Marie JACHIET 1 , Adèle DE MASSON 1 ,
Martine BAGOT 1 and Jean-David BOUAZIZ 1 *
Dermatology Department, and 2 Anatomopathology Department, Saint-Louis Hospital, 1 avenue Claude Vellefaux, 75745 Paris cedex 10,
France. *E-mail: [email protected]
1
Accepted Oct 3, 2019; E-published Oct 3, 2019
Systemic lupus erythematosus (SLE) is a prototypic
autoimmune disease characterized by heterogeneous
clinical and serological manifestations that may affect
any organ (1). Cutaneous lupus erythematosus (CLE) is
an autoimmune disorder occurring independently of or
as a manifestation of SLE, including acute cutaneous
lupus erythematosus, subacute cutaneous lupus, and
chronic cutaneous lupus erythematosus (discoid lupus
erythematosus, lupus erythematosus profundus, chilblain
cutaneous lupus, and lupus tumidus) (2). The prevalence
of vasculitis in SLE is reported to be between 11% and
36%, with a diverse clinical spectrum due to inflam-
matory involvement of vessels of all sizes (3). Pseudo-
erysipelas (PE) is a cutaneous manifestation seen in
periodic fever syndromes, presenting as erythematous
and swollen extremities. We report the case of a man who
developed a PE concomitant with an acute SLE flare.
CASE REPORT
A 42-year-old Guyanese man, diagnosed 12 years ago
with cutaneous, articular and cardiac SLE, who was
not currently undergoing treatment, presented with a
38.5°C fever and symmetrical, painful swelling and
thickening of both legs (Fig. 1A), associated with infil-
trated erythematosus annular lesions of the trunk, back
and face, and atrophic hyperpigmented keratotic lesions
of the face, ears and elbows. Routine biological tests re-
Fig. 1. Symmetrical, painful, swelling and thickening of both legs (A).
Complete remission after steroid treatment (B).
vealed anaemia (10.3 g/dl), neutropenia (1.01 × 10 9 G/l),
lymphopenia (1.05 ×10 9 G/l), and negative CRP (3 mg/l,
normal range < 5 mg/l). Immunological tests revealed
positivity for anti-nuclear antibodies (1/1,600), anti-RNP,
anti-Sm, anti-SSA, and anti-P ribosomes. The C3 and
C4 complement levels were normal. Urine tests revea-
led proteinuria (1.3 g/day). A deep biopsy taken from
the left leg showed a dermo-hypodermal lymphocytic
infiltrate. It involved and surrounded the walls of small
and medium-sized vessels with fibrin deposits in some
of them (Fig. 2). Two other biopsies taken from the back
and the left arm showed dermal lymphocytic infiltrates
with interface dermatitis and mucin deposits in favour of
lupus erythematosus. Renal biopsy showed class II and
V lupus nephritis. Treatment with hydroxychloroquine
and 3-day intravenous injections of 500 mg of methyl-
prednisolone was started, followed by oral prednisone
1 mg/kg/day for one month, which was associated with
the complete remission of the skin lesions and systemic
symptoms (Fig. 1B). Methotrexate (20 mg) once a week
was started as a background treatment for SLE. Progres-
sive tapering of steroids was performed with favourable
clinical evolution during the 6-month follow-up.
DISCUSSION
The originality of this case comes from the associa-
tion of PE and the SLE flare. This is, to the best of our
Fig. 2. Dermo-hypodermal lymphocytic infiltrate surrounding the walls
of small and medium-sized vessels with fibrin deposits in some of them.
This is an open access article under the CC BY-NC license. www.medicaljournals.se/acta
Journal Compilation © 2019 Acta Dermato-Venereologica.
doi: 10.2340/00015555-3334
Acta Derm Venereol 2019; 99: 1295–1296